本文選題：結(jié)核性胸膜炎 + T淋巴細(xì)胞�。� 參考：《山東醫(yī)藥》2017年38期

【摘要】：結(jié)核性胸膜炎是由結(jié)核分枝桿菌抗原引起的T淋巴細(xì)胞介導(dǎo)的胸膜炎癥,其發(fā)病機(jī)制復(fù)雜,至今仍不明確。成熟的T淋巴細(xì)胞是一群高度異質(zhì)性且存在若干亞群的免疫細(xì)胞,研究發(fā)現(xiàn)結(jié)核性胸膜炎患者體內(nèi)存在Ⅰ型T輔助細(xì)胞/Ⅱ型T輔助細(xì)胞免疫應(yīng)答功能紊亂,Th17/Treg失衡及Th9、Th22、黏膜相關(guān)恒定T細(xì)胞免疫增強(qiáng)現(xiàn)象,CD4+IL-27+T細(xì)胞也可通過(guò)干預(yù)T淋巴細(xì)胞亞型的轉(zhuǎn)換來(lái)參與疾病的進(jìn)程。
[Abstract]:Tuberculous pleurisy is a T-lymphocyte mediated pleurisy caused by Mycobacterium tuberculosis antigen. The pathogenesis of tuberculous pleurisy is complex and still unclear. Mature T lymphocytes are a group of highly heterogeneous immune cells with several subsets. It was found that there was a disorder of T helper cell / type 鈪，

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