發(fā)布時(shí)間：2018-04-19 05:08

  本文選題：眩暈 + 磁共振成像�。� 參考：《中風(fēng)與神經(jīng)疾病雜志》2014年05期

【摘要】：目的探討前庭陣發(fā)癥的磁共振表現(xiàn)。方法應(yīng)用三維-磁共振血管成像技術(shù)對(duì)28例前庭陣發(fā)癥(vestibular paroxysmia,VP)患者(VP組)及28例其他眩暈病患者(對(duì)照組)進(jìn)行掃描,評(píng)判所見(jiàn)前庭蝸神經(jīng)周?chē)袩o(wú)神經(jīng)血管交互壓迫(neurovascular cross-compression,NVCC)現(xiàn)象,并記錄責(zé)任血管和責(zé)任血管壓迫神經(jīng)部位至腦干的距離。分別進(jìn)行卡方檢驗(yàn)和t檢驗(yàn)以明確兩組差異之間有無(wú)統(tǒng)計(jì)學(xué)意義。結(jié)果 VP組56耳中存在NVCC現(xiàn)象有35耳,其中血管接觸關(guān)系28耳,推移關(guān)系7耳,對(duì)照組56耳中存在NVCC現(xiàn)象有16耳,均為接觸關(guān)系,未發(fā)現(xiàn)前庭蝸神經(jīng)受血管推移現(xiàn)象,兩組比較差異有統(tǒng)計(jì)學(xué)意義(χ2=16.191,P0.001)。VP組中責(zé)任血管為小腦前下動(dòng)脈25耳(71.4%),小腦后下動(dòng)脈7耳(20%),椎動(dòng)脈3耳(8.6%),對(duì)照組中責(zé)任血管為小腦前下動(dòng)脈9耳(56.3%),小腦后下動(dòng)脈6耳(37.5%),椎動(dòng)脈1耳(6.3%),VP組與對(duì)照組壓迫責(zé)任血管來(lái)源差異無(wú)統(tǒng)計(jì)學(xué)意義(χ2=1.774,P0.05)。NVCC距腦干距離VP組為8.57±5.08 mm,對(duì)照組為8.93±4.64 mm,兩組差異無(wú)統(tǒng)計(jì)學(xué)意義(t=-0.237,P0.05)。VP組NVCC壓迫位置距腦干距離15 mm者為29耳(82.9%),對(duì)照組NVCC壓迫位置距腦干距離15 mm者為12耳(75%),兩組差異無(wú)統(tǒng)計(jì)學(xué)意義(P=0.705,P0.05)。結(jié)論 NVCC在VP患者中發(fā)生率高,壓迫血管以小腦前下動(dòng)脈多見(jiàn),NVCC多發(fā)生在前庭蝸神經(jīng)中樞髓鞘部。
[Abstract]:Objective to investigate the MRI findings of vestibular paroxysma.Methods Twenty eight patients with vestibular paroxysmia VP (VP group) and 28 patients with other vertigo (control group) were scanned by three-dimensional magnetic resonance angiography (MRA). The neurovascular cross-compression (NVCC) phenomenon around the vestibular cochlear nerve was evaluated.The distance between the responsible blood vessel and the responsible blood vessel from the nerve to the brain stem was recorded.Chi-square test and t-test were performed to determine whether there was statistical significance between the two groups.Results there were 35 ears with NVCC in 56 ears of VP group, including 28 ears with vascular contact, 7 ears with NVCC, and 16 ears with NVCC in 56 ears of control group. No vestibular cochlear nerve was found to be vascularized.The difference between the two groups was statistically significant (蠂 ~ 2 / 16.191 / P 0.001N. VP group): 25 ears of anterior inferior cerebellar artery, 71.4% of inferior cerebellar artery, 7 ears of posterior inferior cerebellar artery, 20 ears of posterior inferior cerebellar artery and 8.6% of vertebral artery. In the control group, the responsible vessels were 9 ears of anterior inferior cerebellar artery and 56.33 ears of posterior inferior cerebellar artery, and 6 ears of posterior inferior cerebellar artery.There was no significant difference in the origin of responsible vessels between the VP group and the control group (蠂 ~ 2 / 1.774) (P 0.05). The distance from brain stem to brain stem was 8.57 鹵5.08 mm in VP group and 8.93 鹵4.64 mm in control group. There was no significant difference between the two groups in the distance between NVCC compression position and brainstem. There was no significant difference between the two groups in the distance from the NVCC compression position to the brainstem (15 mm).In the control group, the distance from the NVCC compression position to the brainstem was 15 mm and 12 ears were 75. There was no significant difference between the two groups. There was no significant difference between the two groups (P < 0. 705 and P 0. 05).Conclusion the incidence of NVCC in VP patients is high, and the compression vessels are more common in the central myelin sheath of the vestibulocochlear nerve center than in the anterior inferior cerebellar artery.
【作者單位】： 鄭州大學(xué)第二附屬醫(yī)院神經(jīng)內(nèi)科;暨南大學(xué)醫(yī)學(xué)院;
【基金】：河南省衛(wèi)生廳科技創(chuàng)新人才項(xiàng)目;201004125
【分類(lèi)號(hào)】：R764.3

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