急性胰腺炎胰周血管受累的MRI表現(xiàn)
發(fā)布時間:2018-03-23 00:08
本文選題:急性胰腺炎 切入點:磁共振成像 出處:《川北醫(yī)學院》2014年碩士論文 論文類型:學位論文
【摘要】:目的:探討急性胰腺炎(Acute pancreatitis,,AP)胰周血管受累的磁共振表現(xiàn)、發(fā)生率;胰周血管受累與急性生理慢性健康評分系統(tǒng)Ⅱ(AcutePhysiology And Chronic Healthy Evaluation Ⅱ,APACHⅡ)、磁共振嚴重指數(shù)(Magnetic Resonance severity index,MRSI)的相關性研究。 材料和方法:收集本院2009年8月至2013年8月期間326例AP患者資料,其中男161例,女165例,年齡17-88歲,平均(53±15)歲。所有病例均簽署知情同意書。AP患者均在住院后3天內行腹部平掃加增強檢查,其中122例AP患者采用GE1.5T MR掃描,204例采用GE3.0TMR掃描,序列包括:橫斷面脂肪抑制梯度回波T1加權、橫斷面快速恢復快速自旋回波呼吸門控抑脂T2加權、冠狀面及橫斷面單次激發(fā)快速自旋回波T2加權、單次激發(fā)快速自旋回波胰膽管成像、動態(tài)增強檢查采用抑脂三維肝臟容積快速采集。觀察AP的MRI表現(xiàn),行MRSI評分,0-3分為輕度,4-6分為中度,7-10分為重度。根據(jù)患者臨床資料做APACHⅡ評分,小于8分為輕癥AP,大于(或等于)8分為重癥AP。觀察AP的胰周血管異常表現(xiàn),胰周血管包括脾靜脈、脾動脈、腸系膜上動脈、腸系膜上靜脈、門靜脈、腹腔干、肝總動脈及分支,異常改變包括動脈受侵(炎癥)、假性動脈瘤,靜脈受侵(炎癥)、靜脈血栓、胰源性門靜脈高壓等。用Spearman法統(tǒng)計分析血管并發(fā)癥與MRSI、APACHⅡ評分的相關性。 結果:326例AP患者中,急性間質水腫型AP267例,占81.9%,急性壞死性AP59例,占18.1%。根據(jù)MRSI評分,輕、中、重度分別為38%(124/326)、55%(180/326)、7%(22/326)。16.9%(55/326)的AP患者至少出現(xiàn)一項胰周血管并發(fā)癥。12%(32/267)的急性間質性AP患者發(fā)生胰周血管并發(fā)癥,39%(23/59)的壞死性AP患者發(fā)生胰周血管并發(fā)癥,二者之間的差異具有統(tǒng)計學意義(P<0.05)。輕度、中度、重度AP患者胰周血管并發(fā)癥發(fā)生率分別為3%(4/124)、17%(31/180)、91%(20/22)。其中脾靜脈血栓7例、腸系膜上靜脈血栓5例、脾動脈受侵(炎癥)47例、脾靜脈受侵(炎癥)49例,以上血管并發(fā)癥在MRSI評分輕、中、重度患者中發(fā)生率差異具有統(tǒng)計學意義,并與MRSI評分呈正相關(P<0.05,0.3<r<0.5);腹腔干受侵(炎癥)10例、門靜脈受侵(炎癥)23例、肝總動脈受侵(炎癥)16例、腸系膜上動脈受侵(炎癥)36例、腸系膜上靜脈受侵(炎癥)24例,以上并發(fā)癥在MRSI評分輕、中、重度患者中發(fā)生率差異具有統(tǒng)計學意義,但與MRSI評分無明顯相關性(P<0.05,r<0.3);門靜脈血栓4例、脾動脈假性動脈瘤3例,其發(fā)生率差異不具有統(tǒng)計學意義,與MRSI評分也無明顯相關性(P>0.05,r<0.3)。另有胰源性門靜脈高壓2例。326例AP患者的APACHEⅡ分數(shù)為0-23分,平均5.80±4.620,238例為輕癥AP,88例為重癥AP,14%(34/238)的輕癥患者出現(xiàn)胰周血管并發(fā)癥,24%(21/88)的重癥患者出現(xiàn)胰周血管并發(fā)癥,二者之間的差異具有統(tǒng)計學意義,與急性生理慢性健康評分Ⅱ無相關性(r=0.114,P<0.05)。 結論:AP的胰周血管并發(fā)癥較為常見,包括門靜脈血栓、脾靜脈血栓、腸系膜上靜脈血栓、脾動脈假性動脈瘤、門靜脈炎癥、腸系膜上靜脈炎癥、脾靜脈炎癥、脾動脈炎癥、腸系膜上動脈炎癥、肝總動脈炎癥、腹腔干炎癥、胰源性門靜脈高壓等,其中脾靜脈血栓、脾動靜脈炎癥、腸系膜上靜脈血栓與急性胰腺炎的嚴重程度呈正相關,可以作為一個早期預測AP嚴重程度的指標。急性胰腺炎胰周血管并發(fā)癥與APACHEⅡ評分沒有明顯相關性。
[Abstract]:Objective: To study the acute pancreatitis (Acute, pancreatitis, AP) incidence rate of MRI, peripancreatic vascular involvement; peripancreatic vascular involvement and acute physiology and chronic health evaluation II (AcutePhysiology And Chronic Healthy Evaluation II, APACH II), magnetic resonance (Magnetic Resonance severity index severity index, MRSI) studies.
Materials and methods: collected in our hospital from August 2009 to August 2013 326 cases of AP patients, male 161 cases, female 165 cases, age 17-88 years, average (53 + 15) years old. All patients signed the informed consent.AP patients were in hospital within 3 days of abdominal plain and enhanced scan, 122 cases AP patients with GE1.5T MR scan, 204 cases by GE3.0TMR scan, including sequence: cross sectional gradient echo T1 weighted fat suppression, cross-sectional fast recovery fast spin echo T2 weighted fat suppressed respiratory gating, coronal and axial single shot fast spin echo T2 weighted fast spin echo imaging, dynamic contrast-enhanced examination by fat suppressed 3D liver volume rapid acquisition. To observe the AP MRI expression, MRSI score, 0-3 points to 4-6 points for the mild, moderate, 7-10 were severe. According to the clinical data of patients with APACH score less than 8, divided into mild AP, big In (or equal to) 8 divided into severe AP. observation of peripancreatic vessels AP abnormalities, peripancreatic vessels including splenic vein and splenic artery, superior mesenteric artery, superior mesenteric vein, portal vein, hepatic artery and celiac trunk, branches, abnormal changes including arterial involvement (Yan Zheng), pseudoaneurysm, vein invasion (Yan Zheng), venous thrombosis, pancreatogenous portal hypertension. Spearman with the method of statistical analysis and MRSI correlation of vascular complications, APACH score.
緇撴灉錛
本文編號:1651010
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