沙門氏菌是家禽生產(chǎn)中常見的致病菌,對(duì)肉雞健康危害極大。沙門氏菌感染已經(jīng)成為后抗生素時(shí)代肉雞產(chǎn)業(yè)面臨的重大挑戰(zhàn)。本文的目的是研究德克薩斯側(cè)孢短芽孢桿菌培養(yǎng)物（BT）對(duì)沙門氏菌攻毒肉雞生長(zhǎng)、免疫和血液生化指標(biāo)的影響,并考察德克薩斯側(cè)孢短芽孢桿菌培養(yǎng)物是否具有作為益生菌飼料添加劑的潛力。試驗(yàn)將300只1日齡雄性AA肉雞隨機(jī)分為5個(gè)處理組,每個(gè)處理設(shè)6個(gè)重復(fù),每個(gè)重復(fù)10只雞。處理組包括正對(duì)照（PC,不攻毒并飼喂基礎(chǔ)飼糧）,負(fù)對(duì)照組（NC,攻毒雞白痢沙門氏菌并飼喂基礎(chǔ)飼糧）,抗生素組（AGP,攻毒雞白痢沙門氏菌并飼喂含10 mg/kg吉他霉素抗生素促生長(zhǎng)劑的基礎(chǔ)飼糧）,50 mg/kg BT組（BT50,攻毒雞白痢沙門氏菌并飼喂含50 mg/kg BT的基礎(chǔ)飼糧）和100 mg/kg BT組（BT100,雞白痢沙門氏菌攻毒并飼喂含100 mg/kg BT的基礎(chǔ)飼糧）。攻毒組于7日齡和8日齡分別經(jīng)口灌注雞白痢沙門氏菌菌液一次。42天的試驗(yàn)結(jié)果表明,生長(zhǎng)前期沙門氏菌感染顯著減慢了肉雞的生長(zhǎng)速度,添加AGP和100 mg/kg BT可以部分彌補(bǔ)沙門氏菌的不良影響。但在生長(zhǎng)后期,沙門氏菌、AGP和BT... 

【文章來(lái)源】：中國(guó)農(nóng)業(yè)科學(xué)院北京市

【文章頁(yè)數(shù)】：43 頁(yè)

【學(xué)位級(jí)別】：碩士

【文章目錄】：
摘要
Abstract
Abbreviations
CHAPTER 1 INTRODUCTION
    1.1 BACKROUND
    1.2 AIMS AND OBJECTIVES OF CURRENT STUDY
    1.3 SIGNIFICANCE OF STUDY
CHAPTER 2 LITERATURE REVIEW
    2.1 ANTIMICROBIAL GROWTH PROMOTER (AGP)
    2.2 BIOLOGICAL ROLES OF ANTIMICROBIALS
    2.3 ALTERNATIVES REPLACEMENT FOR ANTIBIOTIC GROWTH PROMOTERS
    2.4 PROBIOTICS AS ALTERNATIVE TO ANTIBIOTICS
    2.5 MECHANISM OF PROBIOTICS ACTION
    2.6 THE GENUS BREVIBACILLUS
    2.7 METABOLISM
    2.8 IDENTIFICATION OF THE BT PEPTIDE
    2.9 BIO-CONTROL AGENT
    2.10 BLOOD BIOCHEMICAL INDICES AND IMMUNE FUNCTIONS
    2.11 SALMONELLOSIS IN POULTRY
        2.11.1 Definition
        2.11.2 Epidemiology
        2.11.3 Host
    2.12 EFFICACY OF PROBIOTICS ON SALMONELLA INFECTED BROILER
CHAPTER 3 PROTECTIVE EFFECT OF BREVIBACILLUS LATEROSPORUS TEXASPORUS CULTURE ON BROILER CHICKENS INFECTED WITH SALMONELLA PULLORUM
    3.1 MATERIALS AND METHODS
        3.1.1 LOCATION AND DURATION
        3.1.2 EXPERIMANTAL DESIGN, ANIMAL AND DIETS
        3.1.3 FEEDING MANAGEMENT
        3.1.4 SALMONELLA CHALLENGE
        3.1.5 GROWTH PERFORMANCE AND DEATH RATE STATISTICS
        3.1.6 SERUM BIOCHEMICAL ANALYSIS
        3.1.7 LIVER HISTOPATHOLOGICAL EXAMINATION
        3.1.8 STATISTICAL ANALYSIS
    3.2 RESULT
        3.2.1 GROWTH PERFORMANCE
        3.2.2 SERUM IMMUNE GLOBAL PROTEIN
        3.2.3 SERUM INFLAMMATORY MARKERS
        3.2.4 LIVER HISTOPATHOLOGICAL ANALYSIS
    3.3 DISCUSSION
CHAPTER 4 CONCLUSION
REFERENCE
ACKNOWLEDGEMENTS
AUTHOR RESUME



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