【摘要】：水稻分蘗是決定植株形態(tài)和產量形成的重要農藝性狀之一。水稻有效分蘗一般由著生于基部非伸長節(jié)間上的分蘗芽伸長生長而來,而著生于伸長節(jié)間上的分蘗由于很難長出稻穗被認為是無效分蘗。為了避免無效分蘗對營養(yǎng)物質的過多消耗,探索抑制伸長節(jié)間上分蘗芽伸長生長的機制,對于改良水稻植株形態(tài)、提高產量形成潛力具有重要意義。前期的研究中,我們鑒定了一個分枝異常突變體abr1,由于伸長節(jié)間上的分蘗芽休眠被打破,從而伸長生長為分蘗,最終表現(xiàn)為分蘗數(shù)增多。在本研究中,我們發(fā)現(xiàn)ABR1調節(jié)上位分蘗芽伸長獨立于獨腳金內脂途徑。ABR1編碼一個胞質定位的蛋白,與兩個同源基因K12A/B分別互作。轉基因株系k12ak12b中也觀察到了類似于abr1突變體的表型,表明K12A/B具有調節(jié)水稻伸長節(jié)間上分蘗芽伸長生長的功能。綜合上述研究結果,初步明確了 ABR1與K12A/B蛋白相互作用調節(jié)伸長節(jié)間上分蘗芽的伸長生長。
[Abstract]:Rice tillering is one of the important agronomic traits that determine the formation of plant morphology and yield. Rice effective tillers are generally derived from the elongation of tiller buds on non-elongated internodes at the base, while tillers on elongated nodes are considered ineffective because of the difficulty in producing rice panicles. In order to avoid excessive consumption of nutrients by ineffective tillers, it is of great significance to explore the mechanism of inhibiting the growth of tiller buds at elongation Internode, which is of great significance for improving the morphology of rice plants and increasing the potential of yield formation. In previous studies, we identified a mutant with abnormal branching, abr1, which was broken by dormancy of the tiller bud at the elongation Internode, and thus grew into a tiller, which eventually showed an increase in the number of tillers. In this study, we found that ABR1 regulates the elongation of superior tiller buds independent of the monopodium internal lipid pathway. ABR1 encodes a cytoplasmic localization protein and interacts with two homologous genes, K12A/B, respectively. Phenotypes similar to those of abr1 mutants were also observed in transgenic line k12ak12b, indicating that K12A/B could regulate the growth of tiller buds on the elongation internodes of rice (Oryza sativa L.). Based on the above results, the interaction between ABR1 and K12A/B protein was used to regulate the growth of tiller buds at elongation Internode.
【學位授予單位】：中國農業(yè)科學院
【學位級別】：博士后
【學位授予年份】：2017
【分類號】：S511

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