加速度造成不對(duì)稱周邊視力喪失的仿真研究
發(fā)布時(shí)間:2018-07-30 07:29
【摘要】:復(fù)合高過載加速度將導(dǎo)致人體不對(duì)稱的周邊視力喪失,會(huì)成為飛行安全的極大隱患;诖,本文提出用數(shù)值仿真的手段探究加速度對(duì)雙眼視力的影響,以期探索復(fù)合加速度造成人體不對(duì)稱周邊視力喪失的力學(xué)機(jī)制。本文首先將計(jì)算機(jī)斷層掃描(CT)的成年人頭骨序列圖像進(jìn)行三維重建,得到含有雙眼眼眶的頭骨仿真模型。再將之前建立的單眼球模型進(jìn)行鏡像,得到雙眼模型,匹配至頭骨模型中,并填充脂肪加以完善。對(duì)該模型分別加載頭-足方向的加速度載荷(Gz)、右-左方向的加速度載荷(Gy)、胸-背方向的加速度載荷(Gx)以及三個(gè)方向的復(fù)合加速度載荷,利用顯式動(dòng)力學(xué)算法,得到視網(wǎng)膜的動(dòng)態(tài)力學(xué)響應(yīng)。仿真研究結(jié)果表明,復(fù)合加速度作用下雙眼應(yīng)變相差25.7%,其分布特征具有明顯差異。本文通過建立雙眼有限元模型,為探索復(fù)合加速度造成不對(duì)稱的周邊視力喪失的機(jī)制性研究提供一種新的手段。
[Abstract]:Compound high overload acceleration will lead to the loss of asymmetric peripheral visual acuity of human body, which will become a great hidden danger of flight safety. Based on this, this paper proposes a numerical simulation method to explore the effect of acceleration on binocular vision, in order to explore the mechanical mechanism of compound acceleration resulting in the loss of human body's asymmetric peripheral visual acuity. In this paper, we first reconstruct the adult skull sequence image of (CT) by computed tomography (CT), and obtain the skull simulation model with binocular orbit. The single eye model was mirrored and the binocular model was matched to the skull model. For the model to load the acceleration load in the head-foot direction respectively, (Gz), acceleration load in the right-left direction, (Gy), acceleration load in the chest and back direction, and the composite acceleration load in the three directions, the explicit dynamic algorithm is used. The dynamic mechanical response of retina was obtained. The simulation results show that the strain difference of binocular strain is 25.7g under the action of compound acceleration, and the distribution characteristics of binocular strain are obviously different. In this paper, a binocular finite element model is established to provide a new method for exploring the mechanism of asymmetric peripheral vision loss caused by composite acceleration.
【作者單位】: 北京航空航天大學(xué)生物與醫(yī)學(xué)工程學(xué)院生物力學(xué)與力生物學(xué)教育部重點(diǎn)實(shí)驗(yàn)室;中國(guó)人民解放軍空軍航空醫(yī)學(xué)研究所;
【基金】:國(guó)家自然科學(xué)基金資助項(xiàng)目(11502013) 國(guó)防基礎(chǔ)科研計(jì)劃資助項(xiàng)目(JCKY2016601B009) 總后重大專項(xiàng)資助項(xiàng)目(AKJ11J002)
【分類號(hào)】:R85
本文編號(hào):2154280
[Abstract]:Compound high overload acceleration will lead to the loss of asymmetric peripheral visual acuity of human body, which will become a great hidden danger of flight safety. Based on this, this paper proposes a numerical simulation method to explore the effect of acceleration on binocular vision, in order to explore the mechanical mechanism of compound acceleration resulting in the loss of human body's asymmetric peripheral visual acuity. In this paper, we first reconstruct the adult skull sequence image of (CT) by computed tomography (CT), and obtain the skull simulation model with binocular orbit. The single eye model was mirrored and the binocular model was matched to the skull model. For the model to load the acceleration load in the head-foot direction respectively, (Gz), acceleration load in the right-left direction, (Gy), acceleration load in the chest and back direction, and the composite acceleration load in the three directions, the explicit dynamic algorithm is used. The dynamic mechanical response of retina was obtained. The simulation results show that the strain difference of binocular strain is 25.7g under the action of compound acceleration, and the distribution characteristics of binocular strain are obviously different. In this paper, a binocular finite element model is established to provide a new method for exploring the mechanism of asymmetric peripheral vision loss caused by composite acceleration.
【作者單位】: 北京航空航天大學(xué)生物與醫(yī)學(xué)工程學(xué)院生物力學(xué)與力生物學(xué)教育部重點(diǎn)實(shí)驗(yàn)室;中國(guó)人民解放軍空軍航空醫(yī)學(xué)研究所;
【基金】:國(guó)家自然科學(xué)基金資助項(xiàng)目(11502013) 國(guó)防基礎(chǔ)科研計(jì)劃資助項(xiàng)目(JCKY2016601B009) 總后重大專項(xiàng)資助項(xiàng)目(AKJ11J002)
【分類號(hào)】:R85
【相似文獻(xiàn)】
相關(guān)期刊論文 前1條
1 徐立;耳脈膊和周邊視力喪失之間的關(guān)系[J];航空軍醫(yī);1997年01期
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